Studies have shown that the inability to effectively absorb manganese may lead to scoliosis in children
October 12, 2018 Source: Xinhua News Agency
Window._bd_share_config={ "common":{ "bdSnsKey":{ },"bdText":"","bdMini":"2","bdMiniList":false,"bdPic":"","bdStyle":" 0","bdSize":"16"},"share":{ }};with(document)0[(getElementsByTagName('head')[0]||body).appendChild(createElement('script')) .src='http://bdimg.share.baidu.com/static/api/js/share.js?v=89860593.js?cdnversion='+~(-new Date()/36e5)];Xinhua News Agency, Washington, October 10 - A new study in the United States says that the inability of the human body to effectively utilize the manganese in the diet caused by genetic variation may cause scoliosis in children. The research paper was published in the new issue of the British journal Nature Communications. Christine Gnette, a senior author of the paper and a professor at Washington University School of Medicine in St. Louis, said the study linked a common disease to a controlled diet. But Gnette also said that people should not blindly supplement manganese, because excessive manganese is harmful to the body.
The study pointed out that children with severe scoliosis had twice the probability of a genetic mutation that would make it difficult for cells to take up and utilize manganese, which is associated with bone growth.
Scoliosis, commonly known as scoliosis, is a three-dimensional deformity of the spine. Mild scoliosis usually has no obvious symptoms. When it is heavier, it will affect the growth and development of young children and make the body deform. When it is serious, it can affect the heart and lung function. Involved in the spinal cord. The pathogenesis of scoliosis in children is unclear.
The team performed genetic testing on 457 children with severe scoliosis and 987 children in the control group. The results showed that the proportion of the variation of the SLC39A8 gene in healthy children was 6%, while that of children with severe scoliosis was 12%. They also performed genetic analysis on an additional 841 children with different degrees of scoliosis and 1,095 healthy children. The results showed that the former group of genes mutated twice as much as the latter group. The researchers also developed zebrafish carrying the above-mentioned genetic defects, which showed motor and skeletal abnormalities, including scoliosis.
Previous studies on the SLC39A8 gene were rare, but studies have shown that it helps cells absorb zinc, iron and manganese. The new study found that after this genetic mutation, the body still can normally absorb zinc and iron, but can not absorb manganese. In children with this genetic variant, the level of manganese in the blood is significantly lower, although it is still within the normal range.
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