Release date: 2016-08-16
Scientists from the University of Cambridge in London and the Royal College of England have successfully suppressed the spread of HIV by freezing small holes in the protein coat. The virus builds infectious DNA through these micropores. Biologists published the report on the study in the journal Nature. HIV is a retrovirus. In order to infect a cell, it must convert the RNA that makes up its gene into DNA. However, scientists don't know how the virus gets the necessary nucleotides—the cornerstone of genetic material. In addition, they are not sure how HIV successfully protects its DNA from the protective system to recognize cells.
The researchers studied the molecular structure of the capsid, a viral coat made up of proteins. In addition, virologists have created a mutant version of HIV to understand how changes in the shell affect its ability to infect. It turns out that there are special micropores in the outer casing that are shaped like a diaphragm. Nucleotides enter the interior through them, including those other foreign molecules responsible for recognizing foreign DNA. When scientists block micropores through hexabromobenzene, the virus loses its ability to replicate itself.
The researchers emphasized that the inhibitors they tested could not penetrate the cells through the plasma membrane. However, they hope to solve this problem in the future and create effective drugs for HIV. In this case, the virus will not be able to multiply and inhibit the activity of the immune system, helping to prevent the development of AIDS.
HIV is a virus that causes defects in the human immune system. In 1983, the human immunodeficiency virus was first discovered in the United States. It is a lentivirus that infects cells of the human immune system and is a type of retrovirus.
Source: Technology World Network
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